Obesity and diabetes are closely linked, often referred to as “diabesity.” Excess adipose tissue particularly visceral fat—plays a key role in the development of insulin resistance, a hallmark of Type 2 diabetes. Adipocytes secrete inflammatory cytokines, such as TNF-α and IL-6, and reduce adiponectin, leading to metabolic dysfunction. Obesity increases free fatty acid levels, disrupts insulin signaling pathways, and contributes to β-cell dysfunction. Individuals with obesity are significantly more likely to develop prediabetes, Type 2 diabetes, and related cardiometabolic conditions. Weight management through dietary intervention, physical activity, behavioral therapy, pharmacological agents, and bariatric surgery has been shown to improve glycemic control and, in some cases, induce diabetes remission. Moreover, public health measures targeting food environments and physical activity are crucial for prevention. Addressing obesity in diabetes care is not only important for glycemic regulation but also for reducing cardiovascular risk, improving mobility, and enhancing quality of life. A multidisciplinary and culturally sensitive approach is vital for long-term success in managing this dual epidemic.